Insulin resistance is one of those terms that is currently circulating widely on social media and in health conversations. Yet, most people lack a clear picture of what it actually means, what causes it, or why it matters beyond the context of diabetes. Understanding it matters because insulin resistance sits at the intersection of stress, emotional eating, weight, and long-term metabolic health, which is precisely why it is clinically relevant for people managing all of these things together.
According to the Public Health Agency of Canada, approximately 11% of Canadian adults aged 20 and older live with diagnosed diabetes, and roughly 201,000 new cases emerge each year. Insulin resistance is the underlying mechanism behind most of these cases, and it often develops silently over years before a formal diagnosis is made. What most people do not realize is that the same chronic stress patterns and emotional eating cycles that drive disordered eating are also among the most significant contributors to insulin resistance, long before diabetes enters the picture.
What Insulin Resistance Actually Is
How Insulin Is Supposed To Work
Insulin is a hormone produced by the pancreas. Insulin’s primary job is to act like a key. When we eat carbohydrate-containing foods, blood glucose levels rise. That is normal and necessary for life, since glucose is our body’s final-stage energy source. The rise in glucose signals the pancreas to release insulin, which then opens the doors of the blood cells, allowing glucose to leave the bloodstream, enter the body’s cells, and nourish vital cells (muscles, fat, liver) throughout the body. Under normal circumstances, this system works efficiently. Blood glucose rises after eating, the pancreas releases insulin, glucose leaves the bloodstream and enters the body’s cells, and blood glucose levels return to baseline.
Insulin resistance occurs when the blood cells (containing glucose) stop responding as efficiently as they should to insulin’s signal. The doors of the blood cells become harder to open, and glucose accumulates in the bloodstream. The pancreas, ever sensitive to the presence of glucose, responds to the problem by producing even more insulin in an effort to fulfill its glucose management role. Without relief, over time, blood glucose remains elevated for longer after meals, and the metabolic conditions that precede type 2 diabetes are in place.
The Cortisol And Glucose Connection
One of the most direct pathways from chronic stress to insulin resistance runs through cortisol. As covered in the post on stress and weight gain, stress keeps cortisol levels persistently elevated. Cortisol’s survival function includes raising blood glucose to provide immediate energy in response to a perceived threat. Research published in Clinical Obesity confirms a bidirectional relationship between cortisol and insulin: Cortisol stimulates food intake (which can raise glucose) while simultaneously impairing insulin signalling, thereby reducing the lowering of glucose.
A Canadian Health Measures Survey study of 3,515 non-diabetic adults, conducted by the Public Health Agency of Canada and Health Canada, found that physically inactive people and those with abdominal weight accumulation had significantly higher odds of insulin resistance. This finding emphasizes the importance of physical activity and reducing abdominal fat. It also highlighted other independent factors that have an insulin-elevating effect, namely chronic stress, which directly results in fat storage, increased appetite and low motivation for physical activity.
Why Stress and Emotional Eating Make It Worse
The Restrict-Binge-Glucose Spike Cycle
Emotional eating driven by stress tends to centre on calorie-dense, high-sugar, high-fat foods, because these foods produce the fastest dopamine response and most reliably quiet the stress response in the short term. The problem is that these foods also produce the most significant blood glucose spikes, requiring the largest insulin responses to manage. For someone whose insulin signaling is already compromised by chronically elevated cortisol, these repeated spikes accelerate the progression of insulin resistance considerably faster than a lower-stress eating pattern would.
A 2025 review in the Journal of Neurochemistry confirms that chronic stress drives disordered eating by disrupting homeostatic balance, with cortisol specifically amplifying hunger signals and promoting intake of high-glucose foods that worsen insulin resistance. Stress eating is not simply a behavioural choice. It is a hormonally driven pattern with direct metabolic consequences.
Understanding why eating happens without physical hunger is a useful starting point for recognizing where stress-driven eating ends and genuine appetite begins, which is one of the first practical steps in interrupting the cycle.
How Poor Sleep Compounds Insulin Resistance
Chronic stress consistently disrupts sleep, and sleep disruption independently worsens insulin resistance through a distinct mechanism. A 2024 systematic review in Endocrine Journal confirmed that acute sleep deprivation significantly affects cortisol levels, producing a blunted morning cortisol response that disrupts glucose regulation from the first hours of the day. Multiple systematic reviews confirm that insufficient sleep increases insulin resistance risk independently of diet and exercise, meaning the stress-sleep disruption-insulin resistance pathway operates even in people who are otherwise eating well.
For people managing chronic stress in high-demand environments, the combination of elevated cortisol, high-glucose stress eating, poor sleep, and reduced physical activity creates compounding conditions in which insulin resistance develops and progresses more quickly than any single factor alone would explain. The post on how anxiety affects gut health explains the related mechanisms by which chronic stress disrupts the digestive system and metabolic regulation.
Stress, Sleep and Emotional Eating Are All Connected. So Is the Support.
If chronic stress and emotional eating are driving metabolic changes for you, Sylvia can help you address the full picture — not just one piece at a time.
What to Do About Insulin Resistance
Dietary adjustments, moderate and regular physical activity, as well as the reduction of mental stress, are the gold standards for the correction of insulin resistance. The mental health and psychotherapy service at Eating Dynamiks & Therapy works with the psychological and emotional dimensions of chronic stress, including how they show up in eating patterns and metabolic health.
The eating management and nutrition counselling service at Eating Dynamiks & Therapy addresses this side of the picture by working with the practical and psychological dimensions of eating in a way that supports metabolic health without adding more food rules to an already strained relationship with food.
For people whose eating patterns, stress levels and relationship with food are all intertwined, the integrated dietitian and psychotherapy service addresses both the nutritional and psychological dimensions simultaneously. You can read more about how this combined approach works in the post on what a holistic approach to mental health and metabolic health looks like in practice.
Ready to Address the Full Picture
If chronic stress, emotional eating and metabolic changes feel connected for you, that connection is real and well-evidenced. Addressing them together, rather than one at a time, is where the most meaningful and durable change tends to happen.
When you are ready to take the next step, get in touch with Sylvia. The first conversation is about understanding what you are dealing with and finding the right starting point.
References
Public Health Agency of Canada. Diabetes in Canada: Data Blog. Public Health Infobase.
https://health-infobase.canada.ca/datalab/diabetes-blog.html
Pan, S.Y., de Groh, M., Aziz, A., & Morrison, H. (2016). Relation of insulin resistance with social-demographics, adiposity and behavioral factors in non-diabetic adult Canadians. Journal of Diabetes and Metabolic Disorders, 15, 65. Public Health Agency of Canada and Health Canada, Ottawa, ON.
Quinkler, M., & Stewart, P.M. (2025). Glucocorticoids and HPA axis regulation in the stress-obesity connection: A comprehensive overview of biological, physiological and behavioural dimensions. Clinical Obesity.
Smith, K.E., et al. (2025). Hunger Games: A Modern Battle Between Stress and Appetite. Journal of Neurochemistry.
Chen, Y., Xu, W., Chen, Y., et al. (2024). The effect of acute sleep deprivation on cortisol level: a systematic review and meta-analysis. Endocrine Journal. Summarised via Vida Integrated Health.
https://thinkvida.com/blog/stress-sleep-cortisol-insulin-resistance/
Canadian Mental Health Association (CMHA). Fast Facts About Mental Health and Mental Illness.
https://cmha.ca/find-info/mental-health/general-info/fast-facts/